Anne-Marie Koop

3 143 SUPPLEMENTAL REFERENCES 1. Cooper G, Satava RM, Harrison CE, Coleman HN. Normal myocardial function and energetics after reversing pressure overload hypertrophy. Am J Physiol Dept. Physiol. Biophys., Mayo Found., Rochester, Minn. 55901; 1974;226:1158–1165. 2. Cooper G 4th, Tomanek RJ, Ehrhardt JC, Marcus ML. Chronic progressive pressure overload of the cat right ventricle. Circ Res United States; 1981;48:488–497. 3. Reibel DK, Uboh CE, Kent RL. Altered coenzyme A and carnitine metabolism in pressure- overload hypertrophied hearts. Am J Physiol - Hear Circ Physiol Dep. Physiol., Thomas Jefferson Univ., Philadelphia, PA 19107; 1983;13:H839–H843. 4. Lauva IK, Brody E, Tiger E, Kent RL, Copper G 4th, Marino TA. Control of myocardial tissue components and cardiocyte organelles in pressure-overload hypertrophy of the cat right ventricle. Am J Anat United States; 1986;177:71–80. 5. Schneider M, Wiese S, Kunkel B, Hauk H, Pfeiffer B. Development and regression of right heart ventricular hypertrophy: Biochemical and morphological aspects. Z Kardiol Zentrum der Pathologie, Universitatsklinik, D-6000 Frankfurt/M; 1987;76:1–8. 6. Olivetti G, Ricci R, Lagrasta C, Maniga E, Sonnenblick EH, Anversa P. Cellular basis of wall remodeling in long-term pressure overload-induced right ventricular hypertrophy in rats. Circ Res Department of Pathology, University of Parma, 43100 Parma; 1988;63:648–657. 7. HungKS, PachecoH, LessinD,JordanK, Mattioli L. Morphometryofrightventricularpapillary muscle in rat during development and regression of hypoxia-induced hypertension. Adv Exp Med Biol K.S. Hung, Department of Anatomy, University of Kansas Medical Center, Kansas City 66103.; 1988;227:337–346. 8. Saito D, Tani H, Kusachi S, Uchida S, Ohbayashi N, Marutani M, Maekawa K, Tsuji T, Haraoka S. Oxygen metabolism of the hypertrophic right ventricle in open chest dogs. Cardiovasc Res D. Saito, Department Cardiovascular Med., Okayama University Med. Sch., Okayama 700, Japan; 1991;25:731–736. 9. Morioka S, Honda M, Ishikawa S, Ishinaga Y, Yano S, Tanaka K, Moriyama K. Changes in contractile and non-contractile proteins, intracellular Ca2+ and ultrastructures during the development of right ventricular hypertrophy and failure in rats. Jpn Circ J S. Morioka, 4th Dept. of Internal Medicine, Shimane Medical University, Izumo, Japan; 1992;56:469–474. 10. Sivitz WI, Lund DD, Yorek B, Grover-McKay M, Schmid PG. Pretranslational regulation of two cardiac glucose transporters in rats exposed to hypobaric hypoxia. Am J Physiol - Endocrinol Metab W.I. Sivitz, Dept. of Internal Medicine, Univ. of Iowa Hospitals and Clinics, Iowa City, IA 52246, United States; 1992;263:E562–E569. 11. Baudet S, Kuznetsov A, Merciai N, Gorza L, Ventura-Clapier R. Biochemical, mechanical and energetic characterization of right ventricular hypertrophy in the ferret heart. J Mol Cell Cardiol S. Baudet, URA CNRS 1340, Lab. de Cardiologie Experimentale, Hopital Laennec, 44035 Nantes Cedex 01, France; 1994;26:1573–1586. 12. Ishikawa K, Hashimoto H, Mitani S, Toki Y, Okumura K, Ito T. Enalapril improves heart failure induced by monocrotaline without reducing pulmonary hypertension in rats: Roles of preserved myocardial creatine kinase and lactate dehydrogenase isoenzymes. Int J Cardiol H. Hashimoto, Department of Internal Medicine, Moriyama Citizen’s Hospital, Nagoya 463, Japan; 1995;47:225–233.

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