Anne-Marie Koop

320 60. Marcus JT, Gan CTJ, Zwanenburg JJM, Boonstra A, Allaart CP, Götte MJW, Vonk- NoordegraafA. InterventricularMechanicalAsynchrony inPulmonaryArterialHypertension. Left-to-Right Delay in Peak Shortening Is Related to Right Ventricular Overload and Left Ventricular Underfilling. J Am Coll Cardiol 2008;51:750–757. 61. Smith BCF, Dobson G, Dawson D, Charalampopoulos A, Grapsa J, Nihoyannopoulos P. Three-dimensional speckle tracking of the right ventricle: Toward optimal quantification of right ventricular dysfunction in pulmonary hypertension. J Am Coll Cardio l 2014;64:41–51. 62. Taylor RR, Covell JW, Sonnenblick EH, Ross J. Dependence of ventricular distensibility on filling of the opposite ventricle. Am J Physiol 1967;213:711–718. 63. BemisCE, SerurJR, BorkenhagenD, Sonnenblick EH, UrschelCW. Influenceofrightventricular filling pressure on left ventricular pressure and dimension. Circ Res 1974;34:498–504. 64. Hardziyenka M, Campian ME, Reesink HJ, Surie S, Bouma BJ, Groenink M, Klemens CA, Beekman L, Remme CA, Bresser P, Tan HL. Right ventricular failure following chronic pressure overload is associated with reduction in left ventricular mass: Evidence for atrophic remodeling. J Am Coll Cardiol Elsevier Inc.; 2011;57:921–928. 65. Bruns DR, Dale Brown R, Stenmark KR, Buttrick PM, Walker LA. Mitochondrial integrity in a neonatal bovine model of right ventricular dysfunction. Am J Physiol - Lung Cell Mol Physiol L.A. Walker, Univ. of Colorado-Denver, Dept. of Medicine, Cardiology, Aurora, United States; 2015;308:L158–L167. 66. Nouette-Gaulain K, Malgat M, Rocher C, Savineau J-P, Marthan R, Mazat J-P, Sztark F. Time course of differential mitochondrial energy metabolism adaptation to chronic hypoxia in right and left ventricles. Cardiovasc Res F. Sztark, Laboratoire d’Anesthésiologie, E.A. Physiologie Mitochondriale, Universite Bordeaux 2, 33076 Bordordeaux, France; 2005;66:132–140. 67. Liu A, Philip J, Vinnakota KC, Bergh F Van den, Tabima DM, Hacker T, Beard DA, Chesler NC. Estrogen maintains mitochondrial content and function in the right ventricle of rats with pulmonary hypertension. Physiol Rep 2017;5:1–12. 68. Broderick TL, KingTM. Upregulation of GLUT-4 in right ventricle of ratswithmonocrotaline- induced pulmonary hypertension. Med Sci Monit T. L. Broderick, Department of Physiology, Midwestern University, Glendale, AZ 85308, United States; 2008;14:BR261–BR264. 69. Enache I, Charles A-L, Bouitbir J, Favret F, Zoll J, Metzger D, Oswald-Mammosser M, Geny B, Charloux A. Skeletal muscle mitochondrial dysfunction precedes right ventricular impairment in experimental pulmonary hypertension. Mol Cell Biochem I. Enache, Service de Physiologie et d’Explorations Fonctionnelles, Pôle de Pathologie Thoracique, Centre Hospitalier Universitaire Strasbourg, Nouvel Hôpital Civil, 67091 Strasbourg, France; 2013;373:161–170. 70. Sutendra G, Dromparis P, Paulin R, Zervopoulos S, Haromy A, Nagendran J, Michelakis ED. A metabolic remodeling in right ventricular hypertrophy is associated with decreased angiogenesis and a transition from a compensated to a decompensated state in pulmonary hypertension. J Mol Med E.D. Michelakis, Department of Medicine, University of Alberta, Edmonton, AB T6G 2B7, Canada; 2013;91:1315–1327. 71. Paulin R, Sutendra G, Gurtu V, Dromparis P, Haromy A, Provencher S, Bonnet S, Michelakis ED. A miR-208-Mef2 axis drives the decompensation of right ventricular function in pulmonary hypertension. Circ Res 2015;116:56–69.

RkJQdWJsaXNoZXIy ODAyMDc0