Aernoud Fiolet

40 Chapter 1 REFERENCES 1. Rochae e Silva M. A brief survey of the history of inflammation. Agents Actions. 1978;8:45–9. 2. Ross R. Atherosclerosis — An Inflammatory Disease. N Engl J Med. 1999;340:115–26. 3. Hansson GK. Inflammation, atherosclerosis, and coronary artery disease. N Engl J Med. 2005;352:1685–95. 4. Falk E, Shah PK. Pathogenesis of Atherothrombosis. Role of Vulnerable, Ruptured, and Eroded Plaques. In: Fuster V, Topol EJ, Nabel EG, editors. Atherothrombosis Coron. Artery Dis. Second edi, Philadelphia, US: Wolters Kluwer Health; 2004, p. 451–61. 5. Libby P, Ridker PM, Hansson GK. Inflammation in Atherosclerosis: From Pathophysiology to Practice. J Am Coll Cardiol. 2009;54:2129–38. 6. DavignonJ,GanzP.Roleof endothelial dysfunction inatherosclerosis.Circulation.2004;109:III27- 32. 7. DiCorleto PE, Fox PL. Vascular Endothelium. In: Fuster V, Topol EJ, Nabel EG, editors. Atherothrombosis Coron. Artery Dis. Second edi, Philadelphia, US: Wolters Kluwer Health; 2004, p. 391–7. 8. Cheng C, Tempel D, van Haperen R, et al. Atherosclerotic lesion size and vulnerability are determined by patterns of fluid shear stress. Circulation. 2006;113:2744–53. 9. Kwak BR, Back M, Bochaton-Piallat ML, et al. Biomechanical factors in atherosclerosis: mechanisms and clinical implications. Eur Heart J. 2014;35:3013–20. 10. Gutiérrez E, Flammer AJ, Lerman LO, et al. Endothelial dysfunction over the course of coronary artery disease. Eur Heart J. 2013;34:3175–81. 11. Williams KJ, Tabas I. The response-to-retention hypothesis of early atherogenesis. Arterioscler Thromb Vasc Biol. 1995;15:551–61. 12. Miller YI, Choi SH, Wiesner P, et al. Oxidation-specific epitopes are danger-associated molecular patterns recognized bypattern recognition receptors of innate immunity. Circ Res. 2011;108:235– 48. 13. Smalley DM, Lin JH, Curtis ML, Kobari Y, Stemerman MB, Pritchard KAJ. Native LDL increases endothelial cell adhesiveness by inducing intercellular adhesion molecule-1. Arterioscler Thromb Vasc Biol. 1996;16:585–90. 14. Li H, Cybulsky MI, Gimbrone Jr MA, Libby P. An atherogenic diet rapidly induces VCAM-1, a cytokine-regulatable mononuclear leukocyte adhesion molecule, in rabbit aortic endothelium. Arterioscler Thromb a J Vasc Biol. 1993;13:197–204. 15. Gosling J, Slaymaker S, Gu L, et al. MCP-1 deficiency reduces susceptibility to atherosclerosis in mice that overexpress human apolipoprotein B. J Clin Invest. 1999;103:773–8. 16. Gerszten RE, Garcia-Zepeda EA, Lim Y-C, et al. MCP-1 and IL-8 trigger firm adhesion of monocytes to vascular endothelium under flow conditions. Nature. 1999;398:718–23. 17. Boring L, Gosling J, Cleary M, Charo IF. Decreased lesion formation in CCR2-/- mice reveals a role for chemokines in the initiation of atherosclerosis. Nature. 1998;394:894–7. 18. Bernelot Moens SJ, Neele AE, Kroon J, et al. PCSK9 monoclonal antibodies reverse the pro- inflammatory profile of monocytes in familial hypercholesterolaemia. EurHeart J. 2017;38:1584– 93. 19. Mackay CR. Chemokines: immunology’s high impact factors. Nat Immunol. 2001;2:95–101. 20. Simionescu M, Simionescu N. Proatherosclerotic events: pathobiochemical changes occurring in the arterial wall before monocyte migration. FASEB J. 1993;7:1359–66.

RkJQdWJsaXNoZXIy ODAyMDc0