60 Chapter 3 three speeches (Mspeech1 = 78.06, SD = 14.85; Mspeech2 = 69.52, SD = 12.91; Mspeech3 = 62.56, SD = 16.92). However, contrary to our first hypothesis there was no significant modulation of this effect by baseline testosterone levels, as indicated by a non-significant Time x Testosterone interaction, Estimate = 0.89(3.50), F(1,50) = 0.06, p = 0.80. The same results were found for the post-assessment SUDs. Time, Estimate = -6.14(1,05), F(1,48) = 34.69, p <.001. Peak SUD scores reduced from speech 1 (M = 78.06, SD = 14.85) to post-assessment (M = 59.67, SD = 18.13); Time x Baseline testosterone Estimate = 3.40(3.09), F(1,48) = 1.21, p = .28. Reductions in fear levels from speech 1 tot post-assessment were not dependent of baseline testosterone. Fear levels males The mixed model analysis for the peak SUDs at the speech exposure session as dependent variable, with baseline testosterone and Time (speech 1, speech 2, speech 3, in the speech exposure session) as predictors, did not show a main effect of Time for males. Fear levels did significantly diminish over speeches, Estimate = -5.81(1.53), F(1,19) = 14.33, p = .001 (Mspeech1 = 66.14, SD = 17.93; Mspeech2 = 61.29, SD = 15.17; Mspeech3 = 54.52, SD = 18.73). Moreover, there was no significant interaction between baseline testosterone levels and Time, Estimate = -.712(7.04), F(1,19) = 1.03, p = .32. Similar effects were found for the post-assessment SUDs: Time, Estimate = -6.70(1.71), F(1,17) = 15.18, p = .001. Fear levels reduced from speech 1 (M = 66.14, SD = 17.93) to post-assessment (M = 46.39, SD = 22.10); Time x Baseline testosterone, Estimate = 0.82(7.47), F(1,16) = 0.01, p = .91. Reductions in fear levels from speech 1 tot post-assessment were not dependent of baseline testosterone. Social anxiety symptom levels females The mixed model analysis for LSAS as dependent variable, with baseline testosterone and Assessment (pre/post) as predictors, showed a main effect of Assessment, again confirming efficacy of exposure, but now with respect to the social anxiety symptom levels, Estimate = -9.73(1.84), F(1,48) = 27.86, p <.001. In specific, LSAS scores reduced from pre to post-assessment (Mpre = 80.33, SD = 22.35; Mpost = 70.74, SD = 24.62). Most critically, an interaction of baseline testosterone and assessment Estimate = 15.76(5.44), F(1,48) = 8.39, p = .006, showed that stronger reductions in symptom levels were related to low baseline testosterone levels. Females show the same results as found in the total sample. We repeated the same mixed models analyses but now with reactive testosterone levels as predictors for females only. Apart from the typical main effect of Assessment (pre, post), Estimate = -9.22(1.92), F(1,47) = 22.99, p <.001, testosterone reactivity did not modulate the effect of Assessment as indicated by a non-significant Time x Assessment
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