Patrick Mulder

226 Chapter 7 REFERENCES 1. Comish; Carlson; Kang; et al. Damage-Associated Molecular Patterns and the Systemic Immune Consequences of Severe Thermal Injury. J. Immunol. 2020, 205, 1189–1197. 2. Jeschke; Chinkes; Finnerty; et al. Pathophysiologic Response to Severe Burn Injury. Ann. Surg. 2008, 248, 387–400. 3. Jeschke; van Baar; Choudhry; et al. Burn Injury. Nat. Rev. Dis. Prim. 2020, 6, 1–25. 4. Eming; Wynn; Martin. Inflammation and Metabolism in Tissue Repair and Regeneration. Science. 2017, 356, 1026–1030. 5. Mulder; Vlig; Fasse; et al. Burn-Injured Skin Is Marked by a Prolonged Local Acute Inflammatory Response of Innate Immune Cells and pro-Inflammatory Cytokines. Front. Immunol. 2022, 13, 1–14. 6. Mulder; Koenen; Vlig; et al. Burn-Induced Local and Systemic Immune Response: Systematic Review and Meta-Analysis of Animal Studies. J. Invest. Dermatol. 2022, 142, 3093-3109.e15. 7. Velnar; Bailey; Smrkolj. The Wound Healing Process: An Overview of the Cellular and Molecular Mechanisms. J. Int. Med. Res. 2009, 37, 1528–1542. 8. Rodrigues; Kosaric; Bonham; et al. Wound Healing: A Cellular Perspective. Physiol. Rev. 2019, 99, 665–706. 9. Koh; DiPietro. Inflammation and Wound Healing: The Role of the Macrophage. Expert Rev. Mol. Med. 2011, 13, e23. 10. Wilgus; Roy; McDaniel. Neutrophils and Wound Repair: Positive Actions and Negative Reactions. Adv. Wound Care 2013, 2, 379–388. 11. Bergquist; Hästbacka; Glaumann; et al. The Time-Course of the Inflammatory Response to Major Burn Injury and Its Relation to Organ Failure and Outcome. Burns 2019, 45, 354–363. 12. Lucas; Waisman; Ranjan; et al. Differential Roles of Macrophages in Diverse Phases of Skin Repair. J. Immunol. 2010, 184, 3964–3977. 13. Mak; Saunders; Jett. T Cell Development, Activation and Effector Functions. In Primer to the Immune Response; Elsevier, 2014; pp. 197–226. 14. Ho; Kupper. T Cells and the Skin: From Protective Immunity to Inflammatory Skin Disorders. Nat. Rev. Immunol. 2019, 19, 490–502. 15. Rendon; Choudhry. Th17 Cells: Critical Mediators of Host Responses to Burn Injury and Sepsis. J. Leukoc. Biol. 2012, 92, 529–538. 16. Sasaki; Zhang; Schwacha; et al. Burn Induces a Th-17 Inflammatory Response at the Injury Site. Burns 2011, 37, 646–651. 17. Hao; Nourbakhsh. Recent Advances in Experimental Burn Models. Biology (Basel). 2021, 10, 526. 18. Abdullahi; Amini-Nik; Jeschke. Animal Models in Burn Research. Cell. Mol. Life Sci. 2014, 71, 3241–3255. 19. Hubrecht; Carter. The 3Rs and Humane Experimental Technique: Implementing Change. Animals 2019, 9, 754. 20. Zomer; Trentin. Skin Wound Healing in Humans and Mice: Challenges in Translational Research. J. Dermatol. Sci. 2018, 90, 3–12. 21. Seok; Warren; Alex; et al. Genomic Responses in Mouse Models Poorly Mimic Human Inflammatory Diseases. Proc. Natl. Acad. Sci. U. S. A. 2013, 110, 3507–3512. 22. Mestas; Hughes. Of Mice and Not Men: Differences between Mouse and Human Immunology. J. Immunol. 2004, 172, 2731–2738. 23. Mathes; Ruffner; Graf-Hausner. The Use of Skin Models in Drug Development. Adv. Drug Deliv. Rev. 2014, 69–70, 81–102. 24. Liu; Rinderknecht; Histing; et al. Establishment of an In Vitro Scab Model for Investigating Different Phases of Wound Healing. Bioengineering 2022, 9, 191. 25. Coolen; Verkerk; Reijnen; et al. Culture of Keratinocytes for Transplantation without the Need of Feeder Layer Cells. Cell Transplant. 2007, 16, 649–661. 26. Bergers; Reijnders; van den Broek; et al. Immune-Competent Human Skin Disease Models. Drug Discov. Today 2016, 21, 1479–1488. 27. Pupovac; Senturk; Griffoni; et al. Toward Immunocompetent 3D Skin Models. Adv. Healthc. Mater. 2018, 7, 1–11. 28. Min; Yun; Lew; et al. The Use of Matriderm and Autologous Skin Graft in the Treatment of Full Thickness Skin Defects. Arch. Plast. Surg. 2014, 41, 330–336.

RkJQdWJsaXNoZXIy MTk4NDMw