44 Chapter 2 REFERENCES 1. Farina; Rosique; Rosique. Curbing Inflammation in Burn Patients. Int. J. Inflam. 2013, 2013, 1–9. 2. Pantalone; Bergamini; Martellucci; et al. The Role of DAMPS in Burns and Hemorrhagic Shock Immune Response: Pathophysiology and Clinical Issues. Review. Int. J. Mol. Sci. 2021, 22, 7020. 3. Eming; Martin; Tomic-Canic. Wound Repair and Regeneration: Mechanisms, Signaling, and Translation. Sci. Transl. Med. 2014, 6, 1–36. 4. Fauerbach; McKibben; Bienvenu; et al. Psychological Distress After Major Burn Injury. Psychosom. Med. 2007, 69, 473–482. 5. Abdullahi; Amini-Nik; Jeschke. Animal Models in Burn Research. Cell. Mol. Life Sci. 2014, 71, 3241–3255. 6. Dahiya. Burns as a Model of SIRS. Front. Biosci. 2009, 14, 4962–4967. 7. Seok; Warren; Alex; et al. Genomic Responses in Mouse Models Poorly Mimic Human Inflammatory Diseases. Proc. Natl. Acad. Sci. U. S. A. 2013, 110, 3507–3512. 8. Zomer; Trentin. Skin Wound Healing in Humans and Mice: Challenges in Translational Research. J. Dermatol. Sci. 2018, 90, 3–12. 9. Page; McKenzie; Bossuyt; et al. The PRISMA 2020 Statement: An Updated Guideline for Reporting Systematic Reviews. BMJ 2021, 372, n71. 10. Fuss; Voloboyeva; Poliovyj. Prognostic Value of Using Neutrophil-Lymphocyte Ratio in Patients with Burn Injury for the Diagnosis of Sepsis and Bacteraemia. Polish J. Surg. 2018, 90, 20–24. 11. Rodrigues; Kosaric; Bonham; et al. Wound Healing: A Cellular Perspective. Physiol. Rev. 2019, 99, 665–706. 12. Velnar; Bailey; Smrkolj. The Wound Healing Process: An Overview of the Cellular and Molecular Mechanisms. J. Int. Med. Res. 2009, 37, 1528–1542. 13. Kotwal; Chien. Macrophage Differentiation in Normal and Accelerated Wound Healing. Macrophages Orig. Funct. Biointervention 2017, 62, 353–364. 14. Guillamat-Prats. The Role of MSC in Wound Healing, Scarring and Regeneration. Cells 2021, 10, 1–15. 15. Hensel; Khattar; Ashton; et al. Characterization of Immune Cell Subtypes in Three Commonly Used Mouse Strains Reveals Gender and Strain-Specific Variations. Lab. Investig. 2019, 99, 93–106. 16. Laggner; Lingitz; Copic; et al. Severity of Thermal Burn Injury Is Associated with Systemic Neutrophil Activation. Sci. Rep. 2022, 12, 1654. 17. Mulder; Vlig; Boekema; et al. Persistent Systemic Inflammation in Patients With Severe Burn Injury Is Accompanied by Influx of Immature Neutrophils and Shifts in T Cell Subsets and Cytokine Profiles. Front. Immunol. 2021, 11, 1–13. 18. Comish; Carlson; Kang; et al. Damage-Associated Molecular Patterns and the Systemic Immune Consequences of Severe Thermal Injury. J. Immunol. 2020, 205, 1189–1197. 19. Jeschke; Gauglitz; Kulp; et al. Long-Term Persistance of the Pathophysiologic Response to Severe Burn Injury. PLoS One 2011, 6, e21245. 20. Marck; Montagne; Tuinebreijer; et al. Time Course of Thrombocytes in Burn Patients and Its Predictive Value for Outcome. Burns 2013, 39, 714–722. 21. Osuka; Ishihara; Shimizu; et al. Natural Kinetics of Blood Cells Following Major Burn: Impact of Early Decreases in White Blood Cells and Platelets as Prognostic Markers of Mortality. Burns 2019, 45, 1901–1907. 22. Sen; Hsei; Tran; et al. Early Clinical Complete Blood Count Changes in Severe Burn Injuries. Burns 2019, 45, 97–102. 23. Manz; Boettcher. Emergency Granulopoiesis. Nat. Rev. Immunol. 2014, 14, 302–314. 24. Heffernan; Monaghan; Thakkar; et al. Failure to Normalize Lymphopenia Following Trauma Is Associated with Increased Mortality, Independent of the Leukocytosis Pattern. Crit. Care 2012, 16, 1–10. 25. Thakkar; Diltz; Drews; et al. Abnormal Lymphocyte Response after Pediatric Thermal Injury Is Associated with Adverse Outcomes. J. Surg. Res. 2018, 228, 221–227. 26. Hu; Wang; Hong; et al. Admission Neutrophil-Lymphocyte Ratio (NLR) Predicts Survival in Patients with Extensive Burns. Burns 2021, 47, 594–600. 27. Italiani; Boraschi. From Monocytes to M1/M2 Macrophages: Phenotypical vs. Functional Differentiation. Front. Immunol. 2014, 5, 1–22. 28. Olingy; San Emeterio; Ogle; et al. Non-Classical Monocytes Are Biased Progenitors of Wound Healing Macrophages during Soft Tissue Injury. Sci. Rep. 2017, 7, 1–16.
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