Patrick Mulder

85 Review Inflammatory Mediators in Animal Burn Models 27. Werner; Grose. Regulation of Wound Healing by Growth Factors and Cytokines. Physiol. Rev. 2003, 83, 835–870. 28. Sawant; Poluri; Dutta; et al. Chemokine CXCL1 Mediated Neutrophil Recruitment: Role of Glycosaminoglycan Interactions. Sci. Rep. 2016, 6, 4–11. 29. Tadie; Bae; Jiang; et al. HMGB1 Promotes Neutrophil Extracellular Trap Formation through Interactions with Toll-like Receptor 4. Am. J. Physiol. - Lung Cell. Mol. Physiol. 2013, 304, 8–12. 30. Hoste; Maueröder; van Hove; et al. Epithelial HMGB1 Delays Skin Wound Healing and Drives Tumor Initiation by Priming Neutrophils for NET Formation. Cell Rep. 2019, 29, 2689-2701.e4. 31. Kapellos; Bonaguro; Gemünd; et al. Human Monocyte Subsets and Phenotypes in Major Chronic Inflammatory Diseases. Front. Immunol. 2019, 10, 1–13. 32. Deshmane; Kremlev; Amini; et al. Monocyte Chemoattractant Protein-1 (MCP-1): An Overview. J. Interf. Cytokine Res. 2009, 29, 313–325. 33. Gschwandtner; Derler; Midwood. More Than Just Attractive: How CCL2 Influences Myeloid Cell Behavior Beyond Chemotaxis. Front. Immunol. 2019, 10, 1–29. 34. Yao; Xu; Jin. Macrophage Polarization in Physiological and Pathological Pregnancy. Front. Immunol. 2019, 10, 1–13. 35. Yunna; Mengru; Lei; et al. Macrophage M1/M2 Polarization. Eur. J. Pharmacol. 2020, 877, 173090. 36. Martinez; Gordon. The M1 and M2 Paradigm of Macrophage Activation: Time for Reassessment. F1000Prime Rep. 2014, 6, 13. 37. Landén; Li; Ståhle. Transition from Inflammation to Proliferation: A Critical Step during Wound Healing. Cell. Mol. Life Sci. 2016, 73, 3861–3885. 38. Kotwal; Chien. Macrophage Differentiation in Normal and Accelerated Wound Healing. Macrophages Orig. Funct. Biointervention 2017, 62, 353–364. 39. Wang; Balaji; Steen; et al. T Lymphocytes Attenuate Dermal Scarring by Regulating Inflammation, Neovascularization, and Extracellular Matrix Remodeling. Adv. Wound Care 2019, 8, 527–537. 40. Entezami; Mosavi. Determination of Lymphocytes Surface Markers in Patients with Thermal Burns and the Influence of Burn Size on Mononuclear Cell Subsets. Med. J. Islam. Repub. Iran 2017, 31, 219–223. 41. Laggner; Lingitz; Copic; et al. Severity of Thermal Burn Injury Is Associated with Systemic Neutrophil Activation. Sci. Rep. 2022, 12, 1654. 42. Toliver-Kinsky; Kobayashi; Suzuki; et al. The Systemic Inflammatory Response Syndrome. In Total Burn Care; Elsevier, 2018; pp. 205–220. 43. Minshawi; Lanvermann; McKenzie; et al. The Generation of an Engineered Interleukin-10 Protein With Improved Stability and Biological Function. Front. Immunol. 2020, 11, 1–18. 44. Ramirez; Patel; Pastar. The Role of TGFβ Signaling in Wound Epithelialization. Adv. Wound Care 2014, 3, 482–491. 45. Murphy; Choileain; Zang; et al. CD4+CD25+ Regulatory T Cells Control Innate Immune Reactivity after Injury. J. Immunol. 2005, 174, 2957–2963. 46. Walker; McKenzie. TH2 Cell Development and Function. Nat. Rev. Immunol. 2018, 18, 121–133. 47. Wang; Wong; Ouyang; et al. Targeting IL-10 Family Cytokines for the Treatment of Human Diseases. Cold Spring Harb. Perspect. Biol. 2019, 11, 1–30. 48. Saraiva; Saraiva; Vieira; et al. Biology and Therapeutic Potential of Interleukin-10. J. Exp. Med. 2020, 217, 1–19. 49. Jarczak; Kluge; Nierhaus. Sepsis—Pathophysiology and Therapeutic Concepts. Front. Med. 2021, 8, 1–22. 50. Barber; Maass; White; et al. Increasing Percent Burn Is Correlated with Increasing Inflammation in an Adult Rodent Model. Shock 2008, 30, 388–393. 51. Jeschke; Mlcak; Finnerty; et al. Burn Size Determines the Inflammatory and Hypermetabolic Response. Crit. Care 2007, 11, 1–11. 52. Yang; Liu; Guo; et al. Investigation and Assessment of Neutrophil Dysfunction Early after Severe Burn Injury. Burns 2021, 47, 1851–1862. 53. Klein; Flanagan. Sex Differences in Immune Responses. Nat. Rev. Immunol. 2016, 16, 626–638. 54. Shansky. Are Hormones a “Female Problem” for Animal Research? Science (80-. ). 2019, 364, 825–826. 55. Boehmer; Goral; Faunce; et al. Age-Dependent Decrease in Toll-like Receptor 4-Mediated Proinflammatory Cytokine Production and Mitogen-Activated Protein Kinase Expression. J. Leukoc. Biol. 2004, 75, 342–349. 56. Renshaw; Rockwell; Engleman; et al. Cutting Edge: Impaired Toll-Like Receptor Expression and Function in Aging. J. Immunol. 2002, 169, 4697–4701. 3

RkJQdWJsaXNoZXIy MTk4NDMw