Ramzi Khalil

Transmembrane Protein 14A protects glomerular filtration barrier integrity 117 6 References 1. Group CW. KDIGO clinical practice guideline for the evaluation and management of chronic kidney disease. Kidney Int Suppl. 2013. 2. Garrett MR, Dene H, Rapp JP. Time-course genetic analysis of albuminuria in Dahl saltsensitive rats on low-salt diet. J Am Soc Nephrol. 2003;14(5):1175-87. 3. Garrett MR, Joe B, Yerga-Woolwine S. Genetic linkage of urinary albumin excretion in Dahl salt-sensitive rats: influence of dietary salt and confirmation using congenic strains. Physiological Genomics. 2006;25(1):39-49. 4. Mehr AP, Siegel AK, Kossmehl P, Schulz A, Plehm R, de Bruijn JA, et al. Early onset albuminuria in Dahl rats is a polygenetic trait that is independent from salt loading. Physiological Genomics. 2003;14(3):209-16. 5. Siegel AK, Kossmehl P, Planert M, Schulz A, Wehland M, Stoll M, et al. Genetic linkage of albuminuria and renal injury in Dahl salt-sensitive rats on a high-salt diet: comparison with spontaneously hypertensive rats. Physiol Genomics. 2004;18(2):218-25. 6. Khalil R, Koop K, Kreutz R, Spaink HP, Hogendoorn PC, Bruijn JA, et al. Increased dynamin expression precedes proteinuria in glomerular disease. J Pathol. 2019;247(2):177-85. 7. Koop K, Eikmans M, Wehland M, Baelde H, Ijpelaar D, Kreutz R, et al. Selective loss of podoplanin protein expression accompanies proteinuria and precedes alterations in podocyte morphology in a spontaneous proteinuric rat model. Am J Pathol. 2008;173(2):315-26. 8. Klammt C, Maslennikov I, Bayrhuber M, Eichmann C, Vajpai N, Chiu EJ, et al. Facile backbone structure determination of human membrane proteins by NMR spectroscopy. Nat Methods. 2012;9(8):834-9. 9. Woo IS, Jin H, Kang ES, Kim HJ, Lee JH, Chang KC, et al. TMEM14A inhibits N-(4hydroxyphenyl)retinamide-induced apoptosis through the stabilization of mitochondrial membrane potential. Cancer Lett. 2011;309(2):190-8. 10. Lee SH, Yoo TH, Nam BY, Kim DK, Li JJ, Jung DS, et al. Activation of local aldosterone system within podocytes is involved in apoptosis under diabetic conditions. Am J Physiol Renal Physiol. 2009;297(5):F1381-90. 11. Zhou LL, Cao W, Xie C, Tian J, Zhou Z, Zhou Q, et al. The receptor of advanced glycation end products plays a central role in advanced oxidation protein products-induced podocyte apoptosis. Kidney Int. 2012;82(7):759-70. 12. Zhou LL, Hou FF, Wang GB, Yang F, Xie D, Wang YP, et al. Accumulation of advanced oxidation protein products induces podocyte apoptosis and deletion through NADPHdependent mechanisms. Kidney Int. 2009;76(11):1148-60. 13. Cardoso VG, Goncalves GL, Costa-Pessoa JM, Thieme K, Lins BB, Casare FAM, et al. Angiotensin II-induced podocyte apoptosis is mediated by endoplasmic reticulum stress/ PKC-delta/p38 MAPK pathway activation and trough increased Na(+)/H(+) exchanger isoform 1 activity. BMC Nephrol. 2018;19(1):179. 14. Tao Y, Yazdizadeh Shotorbani P, Inman D, Das-Earl P, Ma R. Store-operated Ca(2+) entry inhibition ameliorates high glucose and Ang' induced podocyte apoptosis and mitochondria damage. Am J Physiol Renal Physiol. 2023. 15. Yamamoto K, Okabe M, Tanaka K, Yokoo T, Pastan I, Araoka T, et al. Podocytes are lost from glomeruli before completing apoptosis. Am J Physiol Renal Physiol. 2022;323(5):F515-F26. 16. Kimmel CB, Ballard WW, Kimmel SR, Ullmann B, Schilling TF. Stages of embryonic development of the zebrafish. Dev Dyn. 1995;203(3):253-310. 17. Khalil R, Lalai RA, Wiweger MI, Avramut CM, Koster AJ, Spaink HP, et al. Glomerular permeability is not affected by heparan sulfate glycosaminoglycan deficiency in zebrafish embryos. Am J Physiol Renal Physiol. 2019;317(5):F1211-F6.

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