217 leiomyomas and congenital anomalies. Future research should also investigate how UC is affected in different types of adenomyosis and under different types of treatment. Conclusions: Our results confirm differences in uterine movement in adenomyotic versus healthy uteri. This could add to the etiological understanding of clinical symptoms of adenomyosis (i.e. dysmenorrhea and infertility). The notable difference between groups regarding frequency, velocity, amplitude and especially coordination, identifies these features as potential diagnostic or therapeutic targets. Further research into uterine contractility in women with (other) benign uterine disorders, and the effect of treatment on contractility, will hopefully lead to a better understanding of the clinical implications of abnormal uterine contractility. Acknowledgements: We would like to sincerely thank Liselot Wagenaar MD, Blijke Wessel MSc and Cynthia Klaassen MD for their (continued) help with patient inclusions and logistics of the project. Furthermore we continue to be grateful to GE Healthcare (Zipf, Austria) for their support of this research with regards to funding, and for facilitating the use of an ultrasound machine.
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